symptom severity, social impairment and substance abuse; however, no psychophysiological measures were included. These findings are consistent with previous studies which identified as predictors poor role function, earlier onset, and longer duration and greater severity of prodromal symptoms (Amminger et al., 2006; Haroun et al., 2006; Yung et al., 2004). Although less impaired than schizophrenia, CHR patients have generalized neuropsychological deficits (Brewer et al., 2006; Hawkins et al 2004; Woodberry et al., 2010), and verbal memory deficits may be a predictor of psychosis (Brewer et al., 2005; Lencz et al., 2006; Woodberry et al., 2010). A promising line of research has recently implicated various electrophysiologic measures obtained during active and passive auditory paradigms as helpful tools in predicting transition to psychosis (Atkinson et al., 2012; Bodatsch et al., 2011; Frommann et al., 2008; Koh et al., 2011; Shaikh et al., 2012; van der Stelt et al., 2005; van Tricht et al., 2010). However, only smell identification deficits have been shown to discern whom among high-risk cases will specifically develop schizophrenia and its spectrum disorders (Brewer et al., 2003), which is agreement with evidence showing that impairments in odor identification are present before individuals develop psychotic symptoms (Woodberry et al., 2010).
