Alcohol use disorder (AUD) is characterized by compulsive tendencies for alcohol-seeking behavior, excessive alcohol consumption, a vulnerability to craving and relapse from alcohol-associated sensory cues, and persisting alcohol use despite adverse consequences (Cardenas et al., 2018; Ghin et al., 2022). There has been a growing body of evidence indicating that aberrant functional connectivity contributes to dysregulation in multiple brain networks in AUD (Kinreich et al., 2021a; Meyers et al., 2021; Song et al., 2024). Several studies employing fMRI have found AUD associations with hypo-connectivity in frontoparietal executive control networks and hyper-connectivity in salience brain networks compared to healthy controls (Kamarajan et al., 2020; Suk et al., 2021; Weiland et al., 2014). Similarly weak resting state connectivity in left executive control networks has also been reported in opioid use disorder, indicating that this pattern of hypo-connectivity in executive control networks could be a general feature of substance use disorders (Woisard et al., 2021). Numerous investigations into the relationship between AUD on EEG coherence, have reported significantly greater coherence in the theta frequency band compared to non-alcoholic controls indicating aberrant hyper-connectivity between
