Specific patterns of qEEG abnormality associated with specific substance use toxicity such as those found in stimulant abuse or alcohol abuse or with comorbidities such as ADHD (Chabot and Serfontein 1996), PTSD (Huang-Storms et al. 2006) or TBI (Thatcher et al. 1989) suggest underlying brain pathologies that might be amenable to EEG biofeedback that is tailored to the person. These approaches would likely be individualized rather than protocol based, and would be used independently or in conjunction with classic alpha-theta training. By way of example, these could include protocols specific to the qEEG abnormality, such as frontal delta reward to correct the frontal delta deficit in cocaine abuse that Alper (1999) hypothesizes may be related to cocaine sensitization and changes in dopamine transmission. To our knowledge this has never been studied and is clearly a research (not a clinical) recommendation. The qEEG patterns and abnormalities depend significantly on whether the subject is still currently using, the chronicity of use, and the current stage of withdrawal or protracted abstinence. A neurofeedback protocol selected for an individual client with SUD should be
