stress, are inversely correlated with plasma corticosterone levels measured after the last restraint session (Hill et al., 2010b). Importantly, bilateral intra-BLA infusions of the CB1 antagonist, AM251, prior to the last restraint session prevented the normal habituation of the corticosterone response (Hill et al., 2010b). These data link 2-AG-CB1 signaling in the BLA with functional habituation of the neuroendocrine response to repeated homotypic stressors.
