Another interaction effect occurred wherein the presence of either early onset or high baseline drinking (absent the other risk factor) independently increased AUD rates over time, but the combined effects were no greater than those for high baseline drinking only. In this regard, high baseline drinking appears to have subsumed any unique effects attributable to early onset drinking, and the effects of early onset drinking on AUD occurrence in adulthood may at least be partially mediated by drinking levels in early adulthood. It is also interesting to note that there were no interactions with LR. Thus, at least within the SDPS sample, LR appeared to function as a robust predictor of AUDs regardless of the presence or absence of other risk factors.
