Our understanding of the neurophysiological, genetic, and environmental underpinnings of AUD has benefited greatly over the past several decades from the Collaborative Study on the Genetics of Alcoholism (COGA), an interdisciplinary project that has been continuously accumulating multi-modal, multi-generational longitudinal data from families afflicted by AUD (Agrawal et al., 2023; Dick et al., 2023; Johnson et al., 2023). Resting state EEG recordings from COGA participants represent just one of the multiple test batteries from which important discoveries related to functional connectivity mechanisms of AUD have been made (Kinreich, Meyers, et al., 2021; Meyers et al., 2023). Resting state EEG has previously been shown to be a stable individual characteristic and highly heritable (Chorlian et al., 2007; van Beijsterveldt et al., 1996). Researchers using COGA’s resting state EEG data found evidence indicating that increased beta (12-28 Hz) band power could be a risk factor for the development of AUD as it was found not only in individuals with AUD (Rangaswamy et al., 2002), but also in the young offspring of men with AUD (Rangaswamy et al., 2004). Investigations of the effects
