Treated alcoholics also have a higher incidence of psychiatric co-morbidity (Berglund et al., 2006; Di Sclafani et al., 2008; Raimo et al., 1999) than TNAD. Di Sclafani and colleagues (2008) examined a TNAD sample of which the current sample is a subset and found that, in contrast to a long-term abstinent alcohol dependent sample, TNAD did not differ from NAC in psychiatric diagnosis rates. TNAD did have more sub (diagnostic) threshold psychiatric problems than NAC (more symptoms and more abnormal measures of the psychological substrate of psychiatric problems). Moreover, more social and emotional problems, and less engagement in everyday activities are reported for treated samples than TNAD (Lloyd et al., 2004). In addition, large sample studies (Berglund et al., 2006; Raimo et al., 1999) found that alcoholic individuals with no prior treatment history were also more often cohabitating and employed than individuals with treatment histories. Not surprisingly, inadequate diet and nutritional deficiencies are commonly reported in clinical samples of alcoholics. Thiamine deficiency is increasingly identified as an important contributor to alcohol-related brain damage of all kinds (See Martin et al.
