Statistical analyses were conducted using the SPSS statistical package version 19.0 (SPSS, 2010). Demographic data were analyzed using chi-square [sex, race/ethnicity, handedness] and standard analysis of variance (ANOVA) techniques [age and FSIQ]. The following a priori data analysis strategy was employed: three separate 2 (AE) × 2 (ADHD) between-subjects MANCOVAs would be performed on variables from the WISC-IV, D-KEFS, and CANTAB, respectively, using an alpha of p = .05. Pillai’s trace criterion would be used as the omnibus test statistic. We planned to follow up significant interactions of AE and ADHD using a three-step process: (1) an interaction contrast (Maxwell & Delaney, 2004) would determine whether the effect of ADHD in exposed children differed from non-exposed children [(AE+ − AE−) – (ADHD – CON)]; (2) if significant, the interaction contrast would be followed up using simple effects to reveal the source of the interaction; (3) finally, because our research question relates to neuropsychological function of alcohol-exposed children compared to other non-exposed groups, if the source of the interaction was a difference between AE groups, they would be compared independently to
