Oscillations are not epiphenomenological features of brain activity, but have functional relevance. Their synchronicity is a main mode of meso‐ and long‐distance communication between cortical areas and from cortical to subcortical areas (Fries, 2005; Horschig et al., 2015; Schnitzler & Gross, 2005). Coactivation in the form of spike propagation can only take place when brain areas are in synchrony, that is, are both sensitive to input in a depolarizing phase. Different oscillation frequencies subserve different cognitive and affective functions, while sharing the same anatomical network (Akam & Kullmann, 2014; Klimesch, 1996). Such communication has proven essential for executing a wide range of behavioral tasks, may also be affected in behavioral disorders, and may to some extent explain individual differences in behavior (Arns et al., 2013; Doppelmayr et al., 2002; Jenkinson & Brown, 2011). The causal role neural oscillations play in behavioral variation is becoming increasingly clear. For example, blocking beta oscillations (13–30 Hz) in the subthalamic nucleus (STN) of patients with Parkinson's disease during deep brain stimulation treatment can result in relieving bradykinetic symptoms (Engel & Fries, 2010; Swann et
