Results for sedation indicated a main effect of ADH1B gene status (F (1, 62) = 4.23, p < .05; partial η2 = .07), gender (F (1, 62) = 5.90, p < .05; partial η2 = .09). There was also a significant time x gender interaction (F (7, 62) = 3.59, p < .01; partial η2 = .06), with females reporting greater increases in sedation. Follow-up analysis of covariance (ANCOVA) was then used to compare the groups differentiated by allele status at each time point. BrAC for each time point was included as a covariate, except for at baseline, where it was a constant. Results indicated no difference in self-reported sedation across ADH1B groups at baseline (F (1, 66) = 1.10, p = .30; partial η2 = .02). ADH1B*3 participants reported significantly higher sedation at the 15 minute (F (1, 66) = 4.84, p < .05; partial η2 = .07), 60 minute (F (1, 66) = 4.65; p < .05; partial η2 = .07), and 150 minute (F (1, 66) = 5.16; p < .05; partial η2 = .08) time points,
