the bilateral pre- and postcentral regions, right middle frontal gyrus and left inferior frontal gyrus. Furthermore, DTI tractography demonstrated that while alcohol-exposed children displayed a reduction in fiber volume and total number of fibers compared to healthy controls, non-exposed children with ADHD did not significantly differ from either the alcohol-exposed or control groups. Although children with prenatal alcohol exposure may have similar behavioral and cognitive deficits as in ADHD, the underlying neural mechanisms appear to be at least partially distinct.
