etiology, in which the effects of the Maf and AbdB transcription factors on developmental and pathophysiological pathways related to AD are more proximate to the disease endpoint than chloride transport and glycine-related neurochemical systems. (Gaino & Fishell 2002; Pandey 2004; Yamauchi 2005; Lee & Messing 2008; Aguirre et al. 2010; Moonat et al. 2010; Kaun et al. 2011)
