on the fit of the common and independent pathway for OC traits are mixed15,16. An important difference seems to be the number of OC dimensions captured by the scale they used—an independent model fits best when there were more dimensions15, whereas a common model fit best when there were fewer dimensions16. Our finding that shared etiological factors contribute to OC dimensions in youth without being mediated by a latent trait suggests that simply measuring overall OC traits will not uncover the full spectrum of genetic influences on OC dimensions and that OC traits are heterogeneous.
