use, the use of more potent forms of cannabis, and the earlier age of first use has not been accompanied or followed by a commensurate increase in the rates of schizophrenia or an earlier age of onset of the illness. This is difficult to explain—unless perhaps, the increase in the rates of schizophrenia is lagging behind the increase in cannabis use by the general population. It is important to note that schizophrenia is unlikely a homogenous illness. Therefore, it is unlikely for any one environmental factor such as, cannabis exposure or any one gene can account for the disorder. More likely, schizophrenia includes a collection of disorders with some general overlap in manifestations but with diverse pathophysiologies.
