We included patients with GAD as an additional control condition to rule out general effects of anxiety and arousal on α activity. Previous sensor-level analysis of α oscillations revealed no α deficits in GAD (Clancy et al., 2017), which directed us to combine patients with GAD and HCs into one control group in the source-level analysis. Nonetheless, we systematically explored group-specific effects. While the results essentially echoed the main results, we noticed interesting findings of enhanced DMN (mPFC→PCC) α connectivity in GAD at M-RS, in comparison to the PTSD and HC groups. We surmise that this mPFC→PCC hyper-connectivity could heighten DMN functioning in GAD, supporting the hallmark symptoms of self-referential rumination and worry in these patients.
