NoGo conditions and purportedly reflect activation of distinct functional networks involved in response execution and inhibition (Albert et al., 2013, Bekker et al., 2005, Falkenstein, 2006, Jonkman, 2006, Jonkman et al., 2003, Jonkman et al., 2007, Kirmizi-Alsan et al., 2006, Lavric et al., 2004, Nieuwenhuis et al., 2004, Nieuwenhuis et al., 2003, Pandey et al., 2012, Smith et al., 2007, 2008, van Veen and Carter, 2002). The vast majority of studies have implicated N2 and P3 components as reflecting different aspects and temporal features of inhibitory processing in healthy normal subjects as well as reflecting dysfunction in these processes in subjects with several neurological and psychiatric conditions, including alcoholism (Bekker, Kenemans, 2005, Falkenstein, 2006, Kamarajan et al., 2004, 2005a, Kamarajan et al., 2005b, Kirmizi-Alsan, Bayraktaroglu, 2006, Munro et al., 2007, Nieuwenhuis, Yeung, 2004, Nieuwenhuis, Yeung, 2003, Smith et al., 2006, Smith, Johnstone, 2007, 2008).
