et al., 2018), as is often characterised, and directly participates in processes like phagocytosis (Dalton et al., 2018a; Del Giudice & Gangestad, 2018). Our data suggests that genes with immune related functions are involved in the pathogenesis of AN. Further work is now needed to refine whether the immune system is a plausible target for AN that could lead to new treatments. Pathway analysis performed on prioritised genes support these data that genetic risk for AN may exert a functional role in the immune system. The mechanistic action of the immune system in AN pathophysiology remains still largely uncharacterised, however, further experimental exploration of the specific genes prioritised in this study may reveal clinically relevant insights.
