any abnormalities in odor identification, which warrants a more careful review of these results. Relying on the 40-item version of the UPSIT (Doty et al., 1984), Brewer et al. (2003) failed to find SIDs between 59 CHR nonconverters (M = 32.2 ±0.9) or 22 CHR converters (31.2 ±1.6) compared to 31 healthy controls (33.4 ±1.4), but a subgroup of 12 CHR patients who later developed schizophrenia (28.8 ±2.2) differed significantly from all other groups. Whereas Woodberry et al. (2010), using the abbreviated 12-item Brief Smell Identification Test (BSIT) version of the UPSIT, found SIDs for 55 CHR patients (9.9 ±1.5) compared to 34 healthy controls (11.0 ±0.7), apparently the 7 CHR converters (9.7 ±2.2) did not differ from the 44 CHR nonconverters (10.0 ±1.3).2Kamath et al. (2011), a cross-sectional study that also used the Sniffin’ Sticks (Kobal et al., 2000) and therefore addressed the question of pre-conversion differences in at-risk individuals, reported SIDs for 10 CHR (11.40 ±1.07) and 14 genetically at-risk participants (11.93 ±1.49) compared to 17 healthy controls (13.12 ±1.50); however, group differences were not based on raw scores but instead examined via Generalized Linear Latent and Mixed Models (GLLAMM) algorithm. Furthermore, the CHR participants were neither help-
