To check for common findings between our study and previous schizophrenia GWAS studies, we first checked the six SNPs from O'Donovan et al. that remained significant after replication analyses [20]. None of the SNPs were directly genotyped on our platforms, however the three strongest associated SNPs, a well as two of the three others were represented by a proxy SNP with r2≥0.69. Since some of the Munich samples from this study were used in the replication for the O'Donovan et al. paper, we reported association statistics here for the Aberdeen samples only (Table S1). In the Aberdeen dataset, none of the SNPs showed a significant association with schizophrenia.
