been only a single CSD study on anxiety. In a group of individuals with social anxiety, Pause et al. (2010) examined N1 and P3 components of the olfactory potentials in response to chemosensory anxiety signals (i.e., smelling sweat samples donated from either female or male donors) and reported these findings: 1) socially non-anxious females showed much stronger P3-related CSD activation (around 800 ms) at central-parietal regions than males in response to both chemosensory stimuli; and 2) socially anxious female participants showed stronger N1-related CSD activity (435–440 ms) across left and right frontal scalp areas in response to chemosensory anxiety signals than to the control stimuli. However, there are a number of factors that limit the generalizability of these findings: 1) the CSD maps are widely inconsistent across study groups; 2) the N1 sink topography does not correspond with existing findings in the literature; 3) only the source (i.e., positivity) part of the CSD topography is considered and the sink (negativity) has not been explained in the study; and 4) ERP waveforms are filtered with successive 40-Hz and 7 Hz low pass filters at 24 dB, and the noise evident in these (heavily-smoothed) waveforms adversely impact on the CSD transform (order
