excitotoxicity phenotype
Evidence from:
primary |
all sources
Related entities (11)
| Subject | Relation | Object | p-value | Evidence |
|---|---|---|---|---|
| ethanol group | risk_factor_for | excitotoxicity | — | 1 |
| excitotoxicity | risk_factor_for | neuronal loss | — | 1 |
| glutamate | risk_factor_for | excitotoxicity | — | 2 |
| glutathione | protective_against | excitotoxicity | — | 1 |
| NMDAR | risk_factor_for | excitotoxicity | — | 1 |
| NMDA receptor | associated_with | excitotoxicity | — | 1 |
| pyruvate | protective_against | excitotoxicity | — | 1 |
| SLC1A1 | protective_against | excitotoxicity | — | 1 |
| Slc1a2 | protective_against | excitotoxicity | — | 1 |
| Slc1a3 | protective_against | excitotoxicity | — | 1 |
| Slc7a11 | protective_against | excitotoxicity | — | 1 |
Mentioned in (28)
Papers in which this entity is mentioned.
- H-MRS neurometabolite profiles and motor development in school-aged children who are HIV-exposed uninfected: a birth cohort study. (2023)
- The effects of gestational choline supplementation on cerebellar Purkinje cell number in the sheep model of binge alcohol exposure during the first trimester-equivalent. (2022)
- A novel missense variant in ACAA1 contributes to early-onset Alzheimer's disease, impairs lysosomal function, and facilitates amyloid-β pathology and cognitive decline. (2021)
- Simultaneous single cell measurements of intranuclear proteins and gene expression (2021)
- The Role of the Endocannabinoid System and Genetic Variation in Adolescent Brain Development. (2018)
- Perinatal HIV Infection or Exposure Is Associated With Low -Acetylaspartate and Glutamate in Basal Ganglia at Age 9 but Not 7 Years. (2018)
- Machine learning shows association between genetic variability in and cerebral connectivity in preterm infants. (2017)
- Efficient derivation of microglia-like cells from human pluripotent stem cells. (2016)
- Ethanol-Associated Changes in Glutamate Reward Neurocircuitry: A Minireview of Clinical and Preclinical Genetic Findings. (2016)
- Importance of genetics in fetal alcohol effects: null mutation of the nNOS gene worsens alcohol-induced cerebellar neuronal losses and behavioral deficits. (2015)
- Administration of memantine during withdrawal mitigates overactivity and spatial learning impairments associated with neonatal alcohol exposure in rats. (2014)
- Glycine receptors and brain development. (2013)
- Long-lasting neural circuit dysfunction following developmental ethanol exposure. (2013)
- Synaptic effects induced by alcohol. (2013)
- Pilot study of iPS-derived neural cells to examine biologic effects of alcohol on human neurons in vitro. (2012)
- Corpus callosum alterations in very preterm infants: perinatal correlates and 2 year neurodevelopmental outcomes. (2012)
- Prenatal exposure to cigarette smoke or alcohol and cerebellum volume in attention-deficit/hyperactivity disorder and typical development. (2012)
- Administration of memantine during ethanol withdrawal in neonatal rats: effects on long-term ethanol-induced motor incoordination and cerebellar Purkinje cell loss. (2011)
- Fetal alcohol spectrum disorders: experimental treatments and strategies for intervention. (2011)
- The effects of a single memantine treatment on behavioral alterations associated with binge alcohol exposure in neonatal rats. (2011)
- COG1410, an apolipoprotein E-based peptide, improves cognitive performance and reduces cortical loss following moderate fluid percussion injury in the rat. (2010)
- Structural, metabolic, and functional brain abnormalities as a result of prenatal exposure to drugs of abuse: evidence from neuroimaging. (2010)
- Neurotoxic effects and biomarkers of lead exposure: a review. (2009)
- Prenatal NAP+SAL prevents developmental delay in a mouse model of Down syndrome through effects on N-methyl-D-aspartic acid and gamma-aminobutyric acid receptors. (2009)
- Amygdala volume in major depressive disorder: a meta-analysis of magnetic resonance imaging studies. (2008)
- Causes and consequences of methamphetamine and MDMA toxicity. (2006)
- Relationship between availability of NMDA receptor subunits and their expression at the synapse. (2002)
- Mechanisms of alcohol-induced damage to the developing nervous system. (2001)
Merged raw entities (1)
All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.
| Raw name | Type | Papers | Mentions |
|---|---|---|---|
| excitotoxicity | phenotype | 28 | 50 |