apoptosis phenotype

Aliases

OPC death, apoptosis, cell death, natural apoptosis, oligodendrocyte death

Related entities (1)

Mentioned in (100)

Papers in which this entity is mentioned.

• DrBioRight 2.0: an LLM-powered bioinformatics chatbot for large-scale cancer functional proteomics analysis. (2025)
• Massively parallel reporter assay investigates shared genetic variants of eight psychiatric disorders. (2025)
• Functional analysis of cancer-associated germline risk variants. (2025)
• Alcohol Exposure May Increase Prenatal Choline Needs Through Redirection of Choline into Lipid Synthesis Rather than Methyl Donation. (2025)
• Mutations in the bone morphogenetic protein signaling pathway sensitize zebrafish and humans to ethanol-induced jaw malformations. (2025)
• Reduction of APOE accounts for neurobehavioral deficits in fetal alcohol spectrum disorders. (2024)
• ENPP1 Immunobiology as a Therapeutic Target. (2023)
• Embryonic ethanol exposure disrupts craniofacial neuromuscular integration in zebrafish larvae. (2023)
• Human 3D brain organoids: steering the demolecularization of brain and neurological diseases. (2023)
• Gene-alcohol interactions in birth defects. (2023)
• Deterministic evolution and stringent selection during preneoplasia. (2023)
• Alcohol and Immunology: Mechanisms of multi-organ damage. Summary of the 2022 alcohol and Immunology research interest group (AIRIG) meeting. (2023)
• High-throughput detection of craniofacial defects in fluorescent zebrafish. (2023)
• Towards understandings of serine/arginine-rich splicing factors. (2023)
• RNA splicing dysregulation and the hallmarks of cancer. (2023)
• Prenatal alcohol exposure contributes to negative pregnancy outcomes by altering fetal vascular dynamics and the placental transcriptome. (2022)
• At the dawn: cell-free DNA fragmentomics and gene regulation. (2022)
• Explainable multiview framework for dissecting spatial relationships from highly multiplexed data. (2022)
• The pro-apoptotic Bax gene modifies susceptibility to craniofacial dysmorphology following gastrulation-stage alcohol exposure. (2022)
• Transcriptomic analyses of gastrulation-stage mouse embryos with differential susceptibility to alcohol. (2021)
• Loss of tumor protein 53 protects against alcohol-induced facial malformations in mice and zebrafish. (2021)
• Exposure to ethanol leads to midfacial hypoplasia in a zebrafish model of FASD via indirect interactions with the Shh pathway. (2021)
• Macrophage Polarization States in the Tumor Microenvironment. (2021)
• Prenatal alcohol exposure disrupts Sonic hedgehog pathway and primary cilia genes in the mouse neural tube. (2021)
• Simultaneous single cell measurements of intranuclear proteins and gene expression (2021)
• Emerging Roles of SRSF3 as a Therapeutic Target for Cancer. (2020)
• Transcriptome-Wide Regulation of Key Developmental Pathways in the Mouse Neural Tube by Prenatal Alcohol Exposure. (2020)
• Single-cell RNA landscape of intratumoral heterogeneity and immunosuppressive microenvironment in advanced osteosarcoma. (2020)
• Cannabinoids Exacerbate Alcohol Teratogenesis by a CB1-Hedgehog Interaction. (2019)
• Super-enhancers: critical roles and therapeutic targets in hematologic malignancies. (2019)
• Maternal circulating miRNAs that predict infant FASD outcomes influence placental maturation. (2019)
• Designing combination therapies with modeling chaperoned machine learning. (2019)
• Ethanol activates immune response in lymphoblastoid cells. (2019)
• Genome-Informed Targeted Therapy for Osteosarcoma. (2019)
• The role and robustness of the Gini coefficient as an unbiased tool for the selection of Gini genes for normalising expression profiling data. (2019)
• Circular DNA elements of chromosomal origin are common in healthy human somatic tissue. (2018)
• The Cancer Spliceome: Reprograming of Alternative Splicing in Cancer. (2018)
• Quantification of subclonal selection in cancer from bulk sequencing data. (2018)
• Enhanced detection of circulating tumor DNA by fragment size analysis. (2018)
• Diving into the world of alcohol teratogenesis: a review of zebrafish models of fetal alcohol spectrum disorder. (2018)
• Neural correlates of verbal memory in youth with heavy prenatal alcohol exposure. (2018)
• EWS/FLI Confers Tumor Cell Synthetic Lethality to CDK12 Inhibition in Ewing Sarcoma. (2018)
• Universal Patterns of Selection in Cancer and Somatic Tissues. (2017)
• ChimeRScope: a novel alignment-free algorithm for fusion transcript prediction using paired-end RNA-Seq data. (2017)
• Patient-derived xenografts undergo mouse-specific tumor evolution. (2017)
• Mutational landscape of metastatic cancer revealed from prospective clinical sequencing of 10,000 patients. (2017)
• Epigenetics studies of fetal alcohol spectrum disorder: where are we now? (2017)
• PGBD5 promotes site-specific oncogenic mutations in human tumors. (2017)
• Dietary choline levels modify the effects of prenatal alcohol exposure in rats. (2017)
• Fragment Length of Circulating Tumor DNA. (2016)
• Pdgfra and Pdgfrb genetically interact during craniofacial development. (2016)
• Acute alcohol exposure during neurulation: Behavioral and brain structural consequences in adolescent C57BL/6J mice. (2016)
• Small molecule inhibition of cAMP response element binding protein in human acute myeloid leukemia cells. (2016)
• Evolutionary Insights into RNA trans-Splicing in Vertebrates. (2016)
• The Genetics of Fetal Alcohol Spectrum Disorders. (2016)
• Quantifying Clonal and Subclonal Passenger Mutations in Cancer Evolution. (2016)
• The cell cycle regulator CCDC6 is a key target of RNA-binding protein EWS. (2015)
• BET inhibitors induce apoptosis through a MYC independent mechanism and synergise with CDK inhibitors to kill osteosarcoma cells. (2015)
• Oncogenic fusion protein EWS-FLI1 is a network hub that regulates alternative splicing. (2015)
• A germline polymorphism of thymine DNA glycosylase induces genomic instability and cellular transformation. (2014)
• RNA-binding protein RBM20 represses splicing to orchestrate cardiac pre-mRNA processing. (2014)
• A screen of zebrafish mutants identifies ethanol-sensitive genetic loci. (2014)
• MAGeCK enables robust identification of essential genes from genome-scale CRISPR/Cas9 knockout screens. (2014)
• CDK9-mediated transcription elongation is required for MYC addiction in hepatocellular carcinoma. (2014)
• Estimating telomere length from whole genome sequence data. (2014)
• Involvement of p53 in the repair of DNA double strand breaks: multifaceted Roles of p53 in homologous recombination repair (HRR) and non-homologous end joining (NHEJ). (2014)
• Clonal evolution in hematological malignancies and therapeutic implications. (2014)
• EWS-FLI1 utilizes divergent chromatin remodeling mechanisms to directly activate or repress enhancer elements in Ewing sarcoma. (2014)
• Effects of one- and three-day binge alcohol exposure in neonatal C57BL/6 mice on spatial learning and memory in adolescence and adulthood. (2014)
• Prioritizing Potentially Druggable Mutations with dGene: An Annotation Tool for Cancer Genome Sequencing Data. (2013)
• Ewing sarcoma protein: a key player in human cancer. (2013)
• Pdgfra protects against ethanol-induced craniofacial defects in a zebrafish model of FASD. (2013)
• Effects of alcohol on the endocrine system. (2013)
• Commonality in Down and fetal alcohol syndromes. (2013)
• RNA splicing: a new player in the DNA damage response. (2013)
• Different patterns of regional Purkinje cell loss in the cerebellar vermis as a function of the timing of prenatal ethanol exposure in an ovine model. (2013)
• Magnetic resonance microscopy-based analyses of the neuroanatomical effects of gestational day 9 ethanol exposure in mice. (2013)
• LIN28B promotes growth and tumorigenesis of the intestinal epithelium via Let-7. (2013)
• Molecular genetics of B-precursor acute lymphoblastic leukemia. (2012)
• Ethanol-induced face-brain dysmorphology patterns are correlative and exposure-stage dependent. (2012)
• Assessing telomeric DNA content in pediatric cancers using whole-genome sequencing data. (2012)
• The Pediatric Cancer Genome Project. (2012)
• Massive genomic rearrangement acquired in a single catastrophic event during cancer development. (2011)
• Long pre-mRNA depletion and RNA missplicing contribute to neuronal vulnerability from loss of TDP-43. (2011)
• Fetal alcohol spectrum disorders: experimental treatments and strategies for intervention. (2011)
• Fetal alcohol spectrum disorders: research challenges and opportunities. (2011)
• Functional consequences of developmentally regulated alternative splicing. (2011)
• The effects of a single memantine treatment on behavioral alterations associated with binge alcohol exposure in neonatal rats. (2011)
• An integrated ChIP-seq analysis platform with customizable workflows. (2011)
• Imaging the impact of prenatal alcohol exposure on the structure of the developing human brain. (2011)
• Alcohol-induced facial dysmorphology in C57BL/6 mouse models of fetal alcohol spectrum disorder. (2010)
• Structural, metabolic, and functional brain abnormalities as a result of prenatal exposure to drugs of abuse: evidence from neuroimaging. (2010)
• Magnetic resonance microscopy-based analyses of the brains of normal and ethanol-exposed fetal mice. (2010)
• Maternal ethanol consumption alters the epigenotype and the phenotype of offspring in a mouse model. (2010)
• The plausibility of maternal nutritional status being a contributing factor to the risk for fetal alcohol spectrum disorders: the potential influence of zinc status as an example. (2010)
• Insulin-like growth factor-I mitigates motor coordination deficits associated with neonatal alcohol exposure in rats. (2009)
• An RNA code for the FOX2 splicing regulator revealed by mapping RNA-protein interactions in stem cells. (2009)
• An alcohol binding site on the neural cell adhesion molecule L1. (2008)
• 3' end mRNA processing: molecular mechanisms and implications for health and disease. (2008)
• MMEJ repair of double-strand breaks (director's cut): deleted sequences and alternative endings. (2008)

Merged raw entities (8)

All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.