LDL cholesterol phenotype
Evidence from:
primary |
all sources
Related entities (17)
Mentioned in (36)
Papers in which this entity is mentioned.
- Cardiovascular and Metabolic Outcomes in Adults with Fetal Alcohol Spectrum Disorders: A Retrospective Cohort Study. (2026)
- A statistical framework for multi-trait rare variant analysis in large-scale whole-genome sequencing studies. (2025)
- MUSSEL: Enhanced Bayesian polygenic risk prediction leveraging information across multiple ancestry groups. (2024)
- Yield of genetic association signals from genomes, exomes and imputation in the UK Biobank. (2024)
- Untargeted and Targeted Blood Lipidomic Signature Profile of Gestational Alcohol Exposure. (2023)
- A statistical framework for powerful multi-trait rare variant analysis in large-scale whole-genome sequencing studies. (2023)
- Path analysis of the impact of prenatal alcohol on adult vascular function. (2023)
- Development and validation of a trans-ancestry polygenic risk score for type 2 diabetes in diverse populations. (2022)
- AFA: Ancestry-specific allele frequency estimation in admixed populations: The Hispanic Community Health Study/Study of Latinos. (2022)
- Beneficial Effects of Betaine: A Comprehensive Review. (2021)
- Choline metabolome response to prenatal choline supplementation across pregnancy: A randomized controlled trial. (2021)
- The mutational constraint spectrum quantified from variation in 141,456 humans. (2020)
- Polygenic prediction via Bayesian regression and continuous shrinkage priors. (2019)
- Heritability enrichment of specifically expressed genes identifies disease-relevant tissues and cell types. (2018)
- Exploring the phenotypic consequences of tissue specific gene expression variation inferred from GWAS summary statistics. (2018)
- The 3D Genome Browser: a web-based browser for visualizing 3D genome organization and long-range chromatin interactions. (2018)
- Genome-wide association study of alcohol consumption and genetic overlap with other health-related traits in UK Biobank (N=112 117). (2017)
- Extending the MR-Egger method for multivariable Mendelian randomization to correct for both measured and unmeasured pleiotropy. (2017)
- Translating genome-wide association findings into new therapeutics for psychiatry. (2016)
- G = E: What GWAS Can Tell Us about the Environment. (2016)
- Liver PPARα is crucial for whole-body fatty acid homeostasis and is protective against NAFLD. (2016)
- Genetic and environmental influences interact with age and sex in shaping the human methylome. (2016)
- Vitamin D and Reproduction: From Gametes to Childhood. (2015)
- Cytochrome P450 1B1: An unexpected modulator of liver fatty acid homeostasis. (2015)
- DNA methylation signatures link prenatal famine exposure to growth and metabolism. (2014)
- Infant nutrition and later health: a review of current evidence. (2012)
- A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990-2010: a systematic analysis for the Global Burden of Disease Study 2010. (2012)
- Distribution of allele frequencies and effect sizes and their interrelationships for common genetic susceptibility variants. (2011)
- LocusZoom: regional visualization of genome-wide association scan results. (2010)
- New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk. (2010)
- LocusZoom: regional visualization of genome-wide association scan results. (2010)
- Genetics and beyond--the transcriptome of human monocytes and disease susceptibility. (2010)
- Lipid profiles in middle-aged men and women after famine exposure during gestation: the Dutch Hunger Winter Families Study. (2009)
- Potential role for plasma placental growth factor in predicting coronary heart disease risk in women. (2009)
- Repression of smoothened by patched-dependent (pro-)vitamin D3 secretion. (2006)
- C-reactive protein: a critical update. (2003)
Merged raw entities (7)
All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.
| Raw name | Type | Papers | Mentions |
|---|---|---|---|
| ldl cholesterol | phenotype | 17 | 21 |
| ldl | phenotype | 9 | 25 |
| ldl-c | phenotype | 7 | 47 |
| low-density lipoprotein cholesterol | phenotype | 5 | 7 |
| ldl cholesterol | drug | 4 | 5 |
| ldl-c levels | phenotype | 1 | 3 |
| low‑density lipoprotein | phenotype | — | — |