inflammatory response phenotype

Aliases

inflammatory responses

Related entities (5)

Mentioned in (34)

Papers in which this entity is mentioned.

• Chromosomal instability as a driver of cancer progression. (2025)
• Integrated single-cell multiomic profiling of caudate nucleus suggests key mechanisms in alcohol use disorder. (2025)
• Epsin Nanotherapy Regulates Cholesterol Transport to Fortify Atheroma Regression. (2023)
• Maternal ART throughout gestation prevents caudate volume reductions in neonates who are HIV exposed but uninfected. (2023)
• Alcohol and Immunology: Mechanisms of multi-organ damage. Summary of the 2022 alcohol and Immunology research interest group (AIRIG) meeting. (2023)
• Lower Neurocognitive Functioning in HIV-Exposed Uninfected Children Compared With That in HIV-Unexposed Children. (2022)
• Rodent models in placental research. Implications for fetal origins of adult disease. (2022)
• Prenatal Adversity Alters the Epigenetic Profile of the Prefrontal Cortex: Sexually Dimorphic Effects of Prenatal Alcohol Exposure and Food-Related Stress. (2021)
• Childhood Obesity, Cortical Structure, and Executive Function in Healthy Children. (2020)
• Choline, Neurological Development and Brain Function: A Systematic Review Focusing on the First 1000 Days. (2020)
• ASEP: Gene-based detection of allele-specific expression across individuals in a population by RNA sequencing. (2020)
• Effects of chronic intermittent ethanol exposure and withdrawal on neuroblastoma cell transcriptome. (2020)
• Sex Differences in Early Postnatal Microglial Colonization of the Developing Rat Hippocampus Following a Single-Day Alcohol Exposure. (2018)
• The effect of alcohol on the differential expression of cluster of differentiation 14 gene, associated pathways, and genetic network. (2017)
• Fetal sex is associated with maternal stimulated cytokine production, but not serum cytokine levels, in human pregnancy. (2017)
• GeneiASE: Detection of condition-dependent and static allele-specific expression from RNA-seq data without haplotype information. (2016)
• Prenatal alcohol exposure alters steady-state and activated gene expression in the adult rat brain. (2015)
• Dissecting FASD through the global transcriptome. (2015)
• Positively correlated miRNA-mRNA regulatory networks in mouse frontal cortex during early stages of alcohol dependence. (2013)
• Interleukin-6 (IL-6) receptor/IL-6 fusion protein (Hyper IL-6) effects on the neonatal mouse brain: possible role for IL-6 trans-signaling in brain development and functional neurobehavioral outcomes. (2013)
• The contribution of myelin to magnetic susceptibility-weighted contrasts in high-field MRI of the brain. (2012)
• Inflammaging: disturbed interplay between autophagy and inflammasomes. (2012)
• Oncostatin M decreases interleukin-1 β secretion by human synovial fibroblasts and attenuates an acute inflammatory reaction in vivo. (2012)
• Chronic shift-lag alters the circadian clock of NK cells and promotes lung cancer growth in rats. (2012)
• Prenatal alcohol exposure alters the course and severity of adjuvant-induced arthritis in female rats. (2012)
• Long pre-mRNA depletion and RNA missplicing contribute to neuronal vulnerability from loss of TDP-43. (2011)
• The effects of a single memantine treatment on behavioral alterations associated with binge alcohol exposure in neonatal rats. (2011)
• Protection of neurons and microglia against ethanol in a mouse model of fetal alcohol spectrum disorders by peroxisome proliferator-activated receptor-γ agonists. (2011)
• Epigenetics and psychoneuroimmunology: mechanisms and models. (2011)
• Pharmacological treatment of cannabis dependence. (2011)
• MicroRNAs: master regulators of ethanol abuse and toxicity? (2010)
• Novel roles for immune molecules in neural development: implications for neurodevelopmental disorders. (2010)
• Maternal immune activation alters fetal brain development through interleukin-6. (2007)
• SNPs3D: candidate gene and SNP selection for association studies. (2006)

Merged raw entities (2)

All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.