observed even when behavioral differences are not (eg, no performance errors), suggesting that one major strength of ERO data is detection of extremely subtle effects occurring at the neural level, which have important implications for neurocognitive functioning and risk for psychopathology. However, it should be noted that no-go frontal ERO and performance on the GNG task are significantly correlated—suggesting that frontal theta ERO is relevant to task performance, although this is not reflected in a statistically significant behavioral difference among the trauma exposure groups. In the context of the present study, the atypical frontal ERO during the no-go task observed in individuals who were exposed to trauma could be a subtle index of risk for psychopathology and suggests less efficient neural processing during response inhibition, necessitating the use of alternate neural strategies to effectively inhibit their responses in the GNG task. Also of note is the effect of parental history of AUD on the associations of trauma exposure and no-go frontal theta ERO. Given previous evidence that decreased no-go frontal theta ERO is observed in individuals with a family history of AUD, this suggests that the association of sexual assaultive trauma exposure before 10 years with a slower rate of
