Results from the present study also indicated that sexual trauma-related change in frontal no-go theta trajectories influenced risk for young adult AUD symptom count and INT psychopathology, but not CUD symptom count or EXT psychopathology. Taken together, these findings support the hypothesis that early sexual trauma exposure might influence the risk for psychopathology (ie, depression, anxiety, suicidal ideation, or AUDs), in part through neurodevelopmental mechanisms. However, future longitudinal studies are needed to further characterize the potential moderating and/or mediating effects of neurodevelopmental trajectories in the associations of early trauma and later psychopathology. More research is needed to examine other aspects of neural functioning during response inhibition and other aspects of stress-reactivity, including executive control and reward processing.
