For NSSI, results from the discordant twin analysis demonstrated substantially increased risk for twins reporting EAU (OR=7.62). However, odds for NSSI were increased only for DZ twins who reported EAU, but not for MZ twins, thereby suggesting that correlated genes may explain the association between EAU and NSSI. These findings indicate that differences in prevalence rates of NSSI between EAU exposed and unexposed co-twins were likely driven by genes unshared by DZ twins. The nonsignificant association in MZ twins pairs is inconsistent with bivariate twin analyses demonstrating a significant environmental correlation between EAU and NSSI (rE=0.24), which could reflect limited power in the discordant twin analyses. In line with the discordant results, however, the bivariate twin analyses indicated that both EAU and NSSI were influenced by genetic factors, and furthermore, the significant genetic correlation showed that a portion of these genetic influences are shared (rG=0.35). Notably, previous work has found limited evidence for genetic influences on EAU and instead exclusively highlighted the role of unique and shared environmental factors (Richmond-Rakerd et al., 2014). Alternatively, results from the current study, which
