corrected). No effect appeared in the opposite (DMN→VC) direction (ps > 0.12). We further explored whole-brain connectivity with the PCC seed during the M-RS between the PTSD and control groups (Fig. 3). At the familywise threshold, correcting for the 84 Brodmann areas examined (FWE p = 0.0006), only one additional area emerged with attenuated connectivity from the PCC to the right associative auditory cortex (superior temporal gyrus, Brodmann’s area 22; t = −3.93, p = 0.0003) in the PTSD group at MRS. This effect thus aligned with the VC-PCC dysconnectivity in PTSD above.
