Next, simple contrasts of Group for the S-RS revealed reduced α connectivity from the PCC to the VC (PCC→VC) in PTSD (vs controls; t = −2.26, p = 0.027, d = 0.55; Fig. 2A), albeit failing FDR correction. No other effects emerged during this state (ps > 0.34). For the M-RS, the PTSD group (vs controls) demonstrated reduced connectivity within the DMN, including PCC→mPFC (t = −2.82, p = 0.008, FDR p < 0.05, d = 0.69; Fig. 2B) and, at a trend level, mPFC→PCC (t = −1.91, p = 0.064, d = 0.47). The PTSD group demonstrated additional deficits in α connectivity from the VC to both DMN ROIs at M-RS: VC→PCC (t = −3.06, p = 0.004, FDR p < 0.05, d = 0.75) and VC→mPFC (t = −2.05, p = 0.049, d = 0.50; albeit not FDR corrected). No effect appeared in the opposite (DMN→VC) direction (ps > 0.12). We further explored whole-brain connectivity with the PCC seed during the M-RS between the PTSD and control groups (Fig. 3). At the familywise threshold, correcting for the 84
