Across subjects, bilateral occipital (BAs 17-19 and 37), temporal (BA 21), and cingulate (BAs 24, 30) cortex, ventral precuneus (BA 7), thalamus, parahippocampus, amygdala, putamen, caudate, cerebellum, and pons had positive CM (p corr <0.001; Fig. 6); temporal (BAs 20, 22), parietal (BAs 2, 3, and 5), prefrontal (BA 9), cingulate (BA 32), and orbitofrontal (BAs 11 and 47) cortex, and posterior insula had negative CM (p corr <0.001; Fig. 6). The CM in MDTHA, cerebellum and rACC was higher for controls than for cocaine abusers (p corr <0.05; Fig. 5B). There were no statistically significant word-related differences in whole-brain analyses of CM.
