animals, indicating that shLmo3.8 was, at least in some animals, effective in reducing levels of Lmo3 in vivo. Importantly, GFP expression was inversely correlated with Lmo3 expression in shLmo3.8- infected mice (Fig. 3G, R = −0.628, p = 0.029), indicating that higher levels of integrated shLmo3.8 virus correlated with decreased Lmo3 expression in the brains of these mice.