weight from P32-P36 and another increase in weight from P36 to P49 (3.5 g, 19% increase) (Supplemental Figure 1). Mice that received the ethanol binge during adolescence were different from controls in that they did not experience a weight gain until P36, causing the average weight of ethanol treated mice to be significantly less than controls from P32 to P36 (Supplemental Figure 1; *p<0.05 ANOVA with Bonferroni post-tests). By P42, ethanol treated mice recovered and underwent a greater weight gain than controls from P36 to P49 (4.73 g, 31% increase) to arrive at weights by P49 that were not significantly different (Control: 21.7 ± 0.4, Ethanol: 20.23 ± 0.4, p>0.05).
