In conclusion, the results of the current study confirm previous research demonstrating an association between EAU and self-harm. Specifically, the findings suggest that shared genes influence the link between EAU and NSSI, and that there may be a putatively causal link between EAU and SA. This highlights the need for further studies aimed at identifying genetic factors (e.g., predisposition to negative urgency) that contribute to the overlap between EAU and NSSI and through which EAU may causally influence SA (e.g., brain development). The results also suggest that interventions aimed at delaying initiation of alcohol use may be influential in minimizing risk for SA.
