We used three approaches to prioritise genes from these 101 candidates (Supplementary Note; Supplementary Tables 17f, 17g, 18). We identified (i) 32 genes as the single SMR-implicated gene at the locus or through conditional analysis of a locus containing multiple candidates: (ii) 16 genes where the putatively causal eQTLs captured 50% or more of the FINEMAP posterior probability (iii) 29 genes where chromatin conformation analysis (Hi-C analysis of adult and fetal brain) suggested that a promoter of that gene interacted with a putative regulatory element containing a FINEMAP credible SNP28.
